Michael Pusch
Michael Pusch
Istituto di Biofisica, Genova, Italia
Vahvistettu sähköpostiosoite verkkotunnuksessa ge.ibf.cnr.it
Mutation in the neuronal voltage-gated sodium channel SCN1A in familial hemiplegic migraine
M Dichgans, T Freilinger, G Eckstein, E Babini, B Lorenz-Depiereux, ...
The Lancet 366 (9483), 371-377, 2005
Rates of diffusional exchange between small cells and a measuring patch pipette
M Pusch, E Neher
Pflügers Archiv 411 (2), 204-211, 1988
A chloride channel widely expressed in epithelial and non-epithelial cells
A Thiemann, S Gründer, M Pusch, TJ Jentsch
Nature 356 (6364), 57-60, 1992
Mapping the site of block by tetrodotoxin and saxitoxin of sodium channel II
H Terlau, SH Heinemann, W Stühmer, M Pusch, F Conti, K Imoto, S Numa
FEBS letters 293 (1-2), 93-96, 1991
Chloride/proton antiporter activity of mammalian CLC proteins ClC-4 and ClC-5
A Picollo, M Pusch
Nature 436 (7049), 420-423, 2005
Regions involved in the opening of CIC-2 chloride channel by voltage and cell volume
S Gründer, A Thiemann, M Pusch, TJ Jentsch
Nature 360 (6406), 759-762, 1992
Gating of the voltage-dependent chloride channel CIC-0 by the permeant anion
M Pusch, U Ludewig, A Rehfeldt, TJ Jentsch
Nature 373 (6514), 527-531, 1995
Two physically distinct pores in the dimeric CIC-0 chloride channel
U Ludewig, M Pusch, TJ Jentsch
Nature 383 (6598), 340-343, 1996
Heteromultimeric CLC chloride channels with novel properties
C Lorenz, M Pusch, TJ Jentsch
Proceedings of the National Academy of Sciences 93 (23), 13362-13366, 1996
Multimeric structure of ClC‐1 chloride channel revealed by mutations in dominant myotonia congenita (Thomsen).
K Steinmeyer, C Lorenz, M Pusch, MC Koch, TJ Jentsch
The EMBO Journal 13 (4), 737-743, 1994
Molecular determinants of KCNQ (Kv7) K+ channel sensitivity to the anticonvulsant retigabine
A Schenzer, T Friedrich, M Pusch, P Saftig, TJ Jentsch, J Grötzinger, ...
Journal of Neuroscience 25 (20), 5051-5060, 2005
Properties of voltage‐gated chloride channels of the ClC gene family.
TJ Jentsch, W Günther, M Pusch, B Schwappach
The Journal of Physiology 482 (suppl), 19-25, 1995
Myotonia caused by mutations in the muscle chloride channel gene CLCN1
M Pusch
Human mutation 19 (4), 423-434, 2002
The muscle chloride channel ClC-1 has a double-barreled appearance that is differentially affected in dominant and recessive myotonia
C Saviane, F Conti, M Pusch
The Journal of general physiology 113 (3), 457-468, 1999
Mutations in dominant human myotonia congenita drastically alter the voltage dependence of the CIC-1 chloride channel
M Pusch, K Steinmeyer, MC Koch, TJ Jentsch
Neuron 15 (6), 1455-1463, 1995
Surface expression and single channel properties of KCNQ2/KCNQ3, M-type K+ channels involved in epilepsy
M Schwake, M Pusch, T Kharkovets, TJ Jentsch
Journal of Biological Chemistry 275 (18), 13343-13348, 2000
Conservation of chloride channel structure revealed by an inhibitor binding site in ClC-1
R Estévez, BC Schroeder, A Accardi, TJ Jentsch, M Pusch
Neuron 38 (1), 47-59, 2003
Functional and structural conservation of CBS domains from CLC chloride channels
R Estévez, M Pusch, C Ferrer‐Costa, M Orozco, TJ Jentsch
The Journal of physiology 557 (2), 363-378, 2004
Concentration and pH dependence of skeletal muscle chloride channel ClC‐1.
GY Rychkov, M Pusch, DS Astill, ML Roberts, TJ Jentsch, AH Bretag
The Journal of Physiology 497 (2), 423-435, 1996
Permeation and block of the skeletal muscle chloride channel, ClC-1, by foreign anions
GY Rychkov, M Pusch, ML Roberts, TJ Jentsch, AH Bretag
The Journal of general physiology 111 (5), 653-665, 1998
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Artikkelit 1–20